Neuroimagen.com
Un blog con contenidos visuales de neuroimagen
martes, 12 de marzo de 2019
jueves, 21 de abril de 2016
Signo de la golondrina en la Enfermedad de Parkinson
Axial SWI Clinical high resolution 3D-T2*/SWI MRI (Philips ‘PRESTO’ sequence) of a Parkinson's disease patient showing absence of the normal high SWI signal within the nigrosome-1 bilaterally (absent swallow tail sign). Image and text used under creative commons licence CC BY 3.0. Original source: Schwarz ST, Afzal M, Morgan PS et-al. The 'swallow tail' appearance of the healthy nigrosome - a new accurate test of Parkinson's disease: a case-control and retrospective cross-sectional MRI study at 3T. PLoS ONE. 2014;9 (4): e93814. doi:10.1371/journal.pone.0093814 Clinical high resolution 3D-T2*/SWI MRI (Philips ‘PRESTO’ sequence) of a Parkinson's disease patient showing absence of the normal high SWI signal within the nigrosome-1 bilaterally (absent swallow tail sign). Image and text used under creative commons licence. Original source: Schwarz ST, Afzal M, Morgan PS et-al. The 'swallow tail' appearance of the healthy nigrosome - a new accurate test of Parkinson's disease: a case-control and retrospective cross-sectional MRI study at 3T. PLoS ONE. 2014;9 (4): e93814. doi:10.1371/journal.pone.0093814
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Abducens nerve on MRI
Annotated MRI (FIESTA)
Modality: MRI

MRI Axial T2 FIESTA
Annotated high resolution FIESTA sequence through the medulla and pons demonstrates the normal course of the abducens nerve (CN VI) (white arrow) as it ascends from the ponto-medullary junction to Dorello's canal.
Modality: MRI

MRI Axial T2 FIESTA
Annotated high resolution FIESTA sequence through the medulla and pons demonstrates the normal course of the abducens nerve (CN VI) (white arrow) as it ascends from the ponto-medullary junction to Dorello's canal.
sábado, 7 de noviembre de 2015
Resonancia de 7T para detectar Enfermedad de Alzheimer
domingo, 17 de mayo de 2015
Midiendo el volumen del bulbo olfatorio como método diagnóstico para la Enfermedad de Parkinson
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A. Paciente con Atrofia Multisistema, presenta una morfología del bulbo olfatorio normal. B. Paciente con Parkinson, con reducción del volumen del bulbo. |
DOI: http://dx.doi.org/10.1016/j.parkreldis.2015.05.001
sábado, 4 de abril de 2015
Destribución somatotópica de nervios periféricos demostrada por Resonancia Magnética
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Espectro somatotópico de lesiones en distintas afectaciones del nervio ciático demostradas por RM |
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sábado, 28 de mayo de 2011
MR de 7 Tesla en Esclerosis Múltiple
Demencia autoinmune
Neuroimaging in patients with an immunotherapy-responsive cognitive disorder.
Magnetic resonance imaging: yellow arrows indicate areas of abnormality on fluid-attenuated inversion recovery (FLAIR). A, 36-year-old woman (patient 10 in Appendix 1) had fluctuating memory problems and was seropositive for glutamic acid decarboxylase-65 autoantibody. Bilateral hippocampal axial FLAIR abnormality, shown in A1, almost completely resolved after treatment with intravenous (IV) methylprednisolone (A2). B, 51-year-old woman (patient 20 in Appendix 1) had subacute fluctuating memory problems, multifocal neurologic examination findings, and evidence of autoimmunity (IgM antiphospholipid antibody). Symmetric confluent T2 signal abnormality in the white matter of both hemispheres (B1) decreased after treatment with IV methylprednisolone (B2). C, 60-year-old man (patient 41 in Appendix 1) had memory, language, and gait problems and was seropositive for both striational and glutamic acid decarboxylase-65 antibodies. Axial T1 magnetic resonance imaging with contrast demonstrated periventricular vessel enhancement (C1) and resolution after treatment (C2). D, 53-year-old woman (patient 29 in Appendix 1) had memory loss, hallucinations, and subsequent seizure; cerebrospinal fluid protein was elevated (>100 mg/dL), and she was seropositive for thyroid peroxidase antibodies and neuronal and muscle acetylcholine receptor antibodies. Axial FLAIR images show diffusely increased T2 signal in the midbrain (D1), which improved after treatment with IV methylprednisolone (D2). Multiple myeloma was diagnosed 18 months after neurologic presentation.
Positron emission tomographic imaging: Brain reconstructions (brighter color represents regions of hypometabolism) in a 58-year-old man (patient 21 in Appendix 1) who presented with personality change and memory problems and had elevated cerebrospinal fluid protein (>100 mg/dL). Hypometabolism, predominantly frontal and temporal (E1), improved after treatment with IV methylprednisolone (E2).
Single-photon emission computed tomographic brain imaging: Brain neuroimaging in a 35-year-old man (patient 22 in Appendix 1) who presented with vertigo and memory problems, had multiple coexisting autoimmune conditions, and was seropositive for muscle acetylcholine receptor and striational antibodies. Diffuse decrease in uptake in frontotemporoparietal regions (F1) was markedly improved globally after treatment with IV methylprednisolone (F2).
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Encefalitis antiNMDA simulando neuromielitis óptica seronegativa
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